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Ann. N.Y. Acad. Sci., Annals PrePrint, published online ahead of print June 28, 2007 doi: 10.1196/annals.1415.048 Copyright © 2007 by the New York Academy of Sciences description
 4 is Cardioprotective After Myocardial Infarction
1 UCSF, Dept. of Pediatrics and Biochemistry & Biophysics, Gladstone Institute of Cardiovascular Disease, 1650 Owens Street, San Francisco, California, 94158, United States 2 Genetics, Harvard Medical School, 77 Avenue Louis Pasteur, Boston, Massachusetts, 02115, United States 3 Cardiothoracic Surgery, University of Texas Southwestern Medical Center, 5323 Harry Hines Blvd., Dallas, Texas, 75390-8879, United States 4 Cardiothoracic Surgery, University of Texas Southwestern Medical Center, 6000 Harry Hines Blvd., Dallas, Texas, 75390, United States
* To whom correspondence should be addressed. E-mail: dsrivastava{at}gladstone.ucsf.edu. PrePrint Abstract
Heart disease is a leading cause of death in newborns and in adults. Efforts to promote cardiac repair by introduction or recruitment of exogenous stem cells hold promise but typically involve isolation and introduction of autologous or donor progenitor cells. We have found that the G-actin sequestering peptide thymosin Key Words:
thymosin  4, cardiac repair, Integrin Linked Kinase
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